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Exercise protects aged mice against coronary endothelial senescence via FUNDC1-dependent mitophagy

Among the most notable findings was downregulation of pathway annotations related to Alzheimers disease, which the authors interpret as evidence of a shift away from molecular signatures associated with aging and neurodegeneration

Interleukin-1beta Causes Long-Term Potentiation Deficiency in a Mouse Model of Septic Encephalopathy

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